The Glaunsinger Lab

In the photograph, Zoe Davis of the Glaunsinger lab looks at 293T cells in the tissue culture room.

Associate Professor Britt A. Glaunsinger studies how viruses manipulate gene expression in infected host cells. Glaunsinger focuses on the RNA-based regulation and viral factors that target RNA. The Glaunsinger Lab seeks to understand how viral manipulation of host cell gene expression pathways influences viral replication and disease.  Her lab specifically studies gammaherpesviruses, which can cause cancers in people with weakened immune systems. Her work has also shed light on the regulation of gene expression in human cells. 

Associate Professor, Britt Glaunsinger

Britt Glaunsinger is an Associate Professor in the department. She recieved her B.S. in Molecular and Cellular Biology at the University of Arizona in 1995 and her Ph.D. in Molecular Virology and Microbiology at the Baylor College of Medicine in 2001. She worked as a Postdoctoral Fellow in the Hooper Research institute at the University of California in San Francisco for five years before coming the Berkeley in 2006. 

She has published over 25 papers and has recieved many awards including the College of Natural Resources Young Faculty Award (2011), UC Berkeley Prytanean  Faculty Award (2011), W.M. Keck Foundation Distinguished Young Scholars in Medical Research Award (2008-2013), Burroughs Wellcome Investigators in Pathogenesis of Infectious Disease Award (2007-2012) and the Highest Academic Achievement Award from the University of Arizona (1994). She is affiliated with Phi Beta Kappa National Honorary, the American Society for Microbiology, the American Society for Virology, and the RNA Society.

To learn more about the Glaunsinger Lab and their work, visit their website at http://glaunsingerlab.berkeley.edu

 

The Li Ka-Shing Center for Biomedical and Health Sciences
 
 
The Glaunsinger Lab just recently moved into the Li Ka-Shing Center for Biomedical and Health Sciences. Visit glaunsingerlab.berkeley.edu for more information.